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If his serum phospholipid fatty acids are an indicator of his hepatic EFA analysis of fatty acid methyl esters metabolism. then he appears to have an overactivity of the analysis of fatty acid methyl esters delta-5 dehydrogenase (D5DH) and impaired activity of the elongase. One can speculate that consuming GLA and inhibiting D5DH with EPA raised the concentration of DGLA and lowered that of analysis of fatty acid methyl esters AA. An increased ratio of DLGA to AA in phospholipids would encourage the formation of PGE 1, which, as a coronary vasodilator [67], would have a beneficial effect in variant angina. His excessive thirst and dry skin were present despite high levels of circulating n6 EFAs. They improved only when additional EPA was consumed. The role of EPA might have been to correct an n3 deficiency or to modify n6 metabolism by inhibition of D5DH.
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